Infark Miokard - SYI. Home · Documents; Infark Miokard - SYI Patofisiologi Akut Miokard Infark · ALGORITMA INFARK MIOKARD · Algoritma. Download Algoritma Penatalaksanaan Infark Miokard · Infark Miokard Penyimpangan KDM · Download Infark Miokard Penyimpangan KDM · Patofisiologi Akut. Read the latest magazines about Infark and discover magazines on INfark MiOkarD Akut Patofisiologi Dan Penatalaksanaan Penyakit Jantung Koroner.


Author: Lindsey O'Keefe
Country: Antigua
Language: English
Genre: Education
Published: 28 June 2016
Pages: 788
PDF File Size: 23.2 Mb
ePub File Size: 23.62 Mb
ISBN: 301-5-39038-403-4
Downloads: 81020
Price: Free
Uploader: Lindsey O'Keefe


Infark miokard - [Download PPTX Powerpoint]

Symptoms, Causes, Diagnosis, and Treatment Definition and Overview Myocardial infarction, or heart attack in simpler terms, occurs when the heart has been deprived of oxygen usually due to a blockage on one of its arteries, causing the death of the cardiac muscle.

Heart disease is currently one of the leading causes of patofisiologi infark miokard in the United States. According to the Heart Foundation, at leastpeople died of cardiovascular disorder in while aroundpeople have experienced heart attack for the patofisiologi infark miokard time.

A heart attack also occurs every 34 seconds in the country.


It is the only organ that has its own type of muscle, called cardiac muscle, which is patofisiologi infark miokard for pumping volumes of blood 24 hours a day, 7 days a week.

The patofisiologi infark miokard has different types of blood vessels which main function is to distribute blood around the body. These include the arteries, which supply oxygenated blood from the heart to the rest of the body.

The oxygen is necessary for vital metabolic functions of the cells. The heart itself also has arteries such as the aorta, which is the biggest of them all. However, there are cases when they get blocked severely that they prevent patofisiologi infark miokard flow of blood to the body, including the heart.

The person can then go through cardiac arrest, where the heart stops pumping blood, as well as experience stroke, aneurysm weakening of the arteries that can lead to ruptureheart failure, and blood clot formation. Causes of Condition Atherosclerosis — This is a condition characterized by the narrowing of the arteries due to plaque buildup and is normally associated with weakened or damaged arteries.

There is often severe underlying disease e. Although it looks like patofisiologi infark miokard fibrillation, the above image actually shows multifocal atrial tachycardia.

21. Infark Miokard - SYI

Note how there are at least three different P wave configurations! Regular SVT Atrial flutter is common. Other ratios are possible, and sometimes the ratio varies.

This rhythm is often unstable, and the heart may patofisiologi infark miokard in and out of sinus rhythm, or there may be runs of atrial fibrillation. In the above ECG the clue is the rate.

Dysha cardio

A rate of should always engender the suspicion of atrial flutter with 2: Other causes of regular SVT include: The most well-characterised is the Wolff-Parkinson-White syndrome. PR interval under 0. It should be clear that the PR interval will patofisiologi infark miokard be short, and the QRS duration should be prolonged.

Note however that patofisiologi infark miokard everyone with an accessory pathway will conduct all of the time down that pathway.

Pathway Infark Miokard Akut.docx

Accessory pathways are common, estimated to occur in one to three individuals in every thousand. Symptomatic pathways are far less common.

The tachycardias may be due to impulse conduction down via the AV node and back up the accessory pathway commonest, called orthodromic tachycardiathe other way around down accessory pathway, up AV node, termed antidromic tachycardiaor even related to atrial fibrillation.

Distinguishing causes of SVT A few pointers are in order. The important thing to look for is the P wave: A few other hints: Ventricular extrasystoles Because these arise within an ectopic focus within the ventricular muscle, patofisiologi infark miokard QRS complex is wide, bizarre, and unrelated to a preceding P wave.

There is usually a constant relationship timing between the preceding sinus beat and a subsequent ventricular beat, because the preceding patofisiologi infark miokard influences the ectopic focus. The ventricular beat is not usually conducted back into the patofisiologi infark miokard.

What happens to the atrial beat that occurred, or was about to occur when the VE happened?

Related Posts: